By Michael Howell
Local wildlife rehabilitator Judy Hoy is one of the authors cited in a study recently published in the International Journal of Human Nutrition and Functional Medicine which claims to demonstrate that even low doses of glyphosates, a chemical ingredient in many pesticides, can be considered a serious health problem. Lead author of the study is Nancy Swanson, PhD from Abacus Enterprises in Washington state, and Stephanie Seneff, from the Computer Science and Artificial Intelligence Laboratory at Massachusetts Institute of Technology in Boston. The article is entitled: “Evidence that glyphosate is a causative agent in chronic sub-clinical metabolic acidosis and mitochondrial dysfunction.”
The article claims that it is a well-established fact that ingesting large amounts of glyphosate causes metabolic acidosis and other pathophysiologic changes. Clinical signs of acute glyphosate poisoning include severe acidosis determined by low blood pH, hyperkalemia, hypernatremia, raised creatinine and blood urea levels, hypotension, hypoxemia and reduced serum bicarbonate. Severe poisoning causes dehydration, pneumonitis, oliguria, altered level of consciousness, hepatic dysfunction, pulmonary edema and dysrhythmias 1, 2, 3.
The authors go on to claim, however, that available scientific reports and records from the CDC examined and compared in their study provide overwhelming “circumstantial evidence” that ingestion of glyphosates in low doses also has serious health effects which are being overlooked in toxicology evaluations and public policy.
“How much evidence is needed?” they ask in the article.
“Taken together, this evidence suggests that glyphosate, in the doses equivalent to allowed residues in food ingested over a long period of time, causes a low-grade, chronic acidosis as well as mitochondrial dysfunction,” states the study.
They also provide evidence from the literature supporting the biochemical pathways whereby this occurs by extracting the reports for symptoms and diseases associated with glyphosate from the U.S. Food and Drug Administration’s Adverse Event Reporting System database. These are compared to the symptoms and diseases reported in the database for drugs that are known to cause mitochondrial dysfunction. They call the results “startlingly consistent.”
Finally, they hypothesize that many modern diseases are primarily acquired mitochondrial disorders caused by chemical pesticides, pharmaceutical drugs, food additives and industrial chemicals.